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Additional info for Advances in lipid research. Volume 11
The apoproteins of the HDL have not been specifically identified. However, cholesterol Peak III Peak II FIG. 24. Erectron micrographs of LDL subfractions from cholesterol-fed guinea pigs. Parts A-D as indicated in the figure. Peaks refer to those shown in Fig. 23. 59,500 x; inset in C, 153,000 X. From Sardet et al. (1972a). Pea k I Whole Chol. l 38 JOHN A. GLOMSET AND KAARE R. 31 20 40 40 100 Data from Sardet et ale (1972a). PL = phospholipid. e Peaks correspond to peaks in Fig. 23. d Whole LDL from animals on control diet.
35 Lecithin: Cholesterol Acyltransferase the guinea pig, and in this species many of the abnormalities induced by the dietary cholesterol resemble those in familial LCAT deficiency. , 1967). The lipoprotein abnormalities will be described below; possible causes and effects of the abnormalities will be discussed in Section V,B. 44) and migrate more slowly on electrophoresis ("f3" or "slow pre-f3" mobility). These features are reminiscent of those already described for the VLDL of patients with familial LCAT deficiency.
L-~------LT---__, 100 400 500 FIG. 14. Effect of diet on the HDL of a patient with familial LCAT deficiency. Patient M. R. was successively fed a diet containing (A) 35% of the total calories as fat, (B) an isocaloric, fat-free diet, and (C) a diet containing 35% of the total calories as low cholesterol fat. At the end of each dietary period (2, 5, and 4 days respectively) the HDL were prepared by ultracentrifugation and subfractionated by gel filtration on Sephadex G 200. From Glomset et ale (1973b).