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By Rodolfo Paoletti; David Kritchevsky

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The apoproteins of the HDL have not been specifically identified. However, cholesterol Peak III Peak II FIG. 24. Erectron micrographs of LDL subfractions from cholesterol-fed guinea pigs. Parts A-D as indicated in the figure. Peaks refer to those shown in Fig. 23. 59,500 x; inset in C, 153,000 X. From Sardet et al. (1972a). Pea k I Whole Chol. l 38 JOHN A. GLOMSET AND KAARE R. 31 20 40 40 100 Data from Sardet et ale (1972a). PL = phospholipid. e Peaks correspond to peaks in Fig. 23. d Whole LDL from animals on control diet.

35 Lecithin: Cholesterol Acyltransferase the guinea pig, and in this species many of the abnormalities induced by the dietary cholesterol resemble those in familial LCAT deficiency. , 1967). The lipoprotein abnormalities will be described below; possible causes and effects of the abnormalities will be discussed in Section V,B. 44) and migrate more slowly on electrophoresis ("f3" or "slow pre-f3" mobility). These features are reminiscent of those already described for the VLDL of patients with familial LCAT deficiency.

L-~------LT---__, 100 400 500 FIG. 14. Effect of diet on the HDL of a patient with familial LCAT deficiency. Patient M. R. was successively fed a diet containing (A) 35% of the total calories as fat, (B) an isocaloric, fat-free diet, and (C) a diet containing 35% of the total calories as low cholesterol fat. At the end of each dietary period (2, 5, and 4 days respectively) the HDL were prepared by ultracentrifugation and subfractionated by gel filtration on Sephadex G 200. From Glomset et ale (1973b).

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